API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine

Model NO.
Dsung-Capecitabine
Mf
C15h22fn3o6
MW
359.35
CAS No.
154361-50-9
Shelf Life
2 Years
Boiling Point
437.874 C at 760 Mmhg
Refractive Index
1.7
Melting Point
110-121 C
Density
1.49
Application
Anti-Cancer
Assay
HPLC 99%
Test Method
HPLC
Specific
COA
Product Name
Capecitabine
Transport Package
Negotiable
Specification
99%
Trademark
D-Sung
Origin
Shaanxi
Production Capacity
3 Ton/Month
Reference Price
$ 180.00 - 360.00
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Product Description

API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine
API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine
 
Product Description

API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine

Product Details

API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine

Product Name Capecitabine
Appearance white to off-white powder
CAS No. 154361-50-9
MF C15H22FN3O6
MW 359.35
Capecitabine is an anti-metabolite fluoropyrimidine deoxynucleoside carbamate that can be converted into 5-FU in the body.It was developed by Roche and its trade name is Xeloda.It can inhibit cell division and interfere with RNA and protein.(protein) synthesis.Indicated for the further treatment of advanced primary or metastatic breast cancer that is refractory to paclitaxel and chemotherapy regimens including anthracyclines.Mainly used for the treatment of advanced primary or metastatic breast cancer, rectal cancer, colon cancer and gastric cancer.
Application & Function

API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine Capecitabine is an orally-administered chemotherapeutic agent used in the treatment of metastatic breast and colorectal cancers.Capecitabine is a prodrug, that is enzymatically converted to fluorouracil (antimetabolite) in the tumor, where it inhibits DNA synthesis and slows growth of tumor tissue.
Capecitabine is rapidly absorbed by the intestinal mucosa after oral administration, and then converted into an inactive intermediate 5'-deoxy-5' fluorocytidine in the liver by carboxyl esterase, followed by cytidine removal from liver and tumor tissues.The action of the aminoase is converted to 5'-deoxy-5' fluorouridine and finally catalyzed by thymidine phosphorylase to fluorouracil (5-FU) in tumor tissues.  Clinical studies have shown that the concentration of 5-FU in tumor tissue after oral administration is significantly higher than that of blood and muscle. The efficacy of tumors in a variety of animals was significantly higher than 5-FU.

Specification

API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine API Capecitabine Powder Purity Pharmaceutical Capecitabine Raw Material Powder Capecitabine

 

 

Pharmaceutical Intermediate

PNEUTEC.IT, 2023